Technology will kill the 9-to-5 work week, says Richard Branson
The billionaire entrepreneur predicts the rise of technology will soon force society to rethink the modern work week.
- Branson made the argument in a recent blog post published on the Virgin website.
- The 40-hour work week stems from labor laws created in the early 20th century, and many have said this model is becoming increasingly obsolete.
- The average American currently works 47 hours per week, on average.
Have you ever had a 'case of the Mondays,' or remarked that you can't believe it's 'hump day' already, or said TGIF to a coworker at the end of a regular, 9-to-5 week? The chances are that, as A.I. continues to improve, you'll soon be able to retire these awful sayings because the work week is poised to undergo a major transformation.
That's the idea behind a recent blog post written by entrepreneur Richard Branson, who's established flexible work arrangements at his company Virgin Management.
"The idea of working five days a week with two day weekends and a few weeks of holiday each year has become ingrained in society. But it wasn't always the case, and it won't be in the future," Branson wrote.
"As Google's Larry Page and others have said, the amount of jobs available for people is going to decrease as technology progresses. New innovations will drive industries forward, but they will also reduce our reliance on people power," Branson wrote. "Ideas such as driverless cars and more advanced drones are becoming a reality, and machines will be used for more and more jobs in the future. Even pilot-less planes will be become the reality in the not too distant future."
Brief history of the modern work week
The 9-to-5, Monday-to-Friday work week might sound tiring and overly regimented to us now. But establishing this relatively new labor model was considered a victory for workers in the 19th and 20th centuries who, in various movements worldwide, organized to demand better labor conditions and regulation. In the early 20th century, for instance, many Americans worked brutally long hours in terrible conditions before Congress passed the Fair Labor Standards Act in 1938, which essentially created the modern American work week that we know today.
But recently some have questioned the utility of the model, citing how technology enables people to work remotely, jobs are different than they once were, and studies that suggest a shorter workday, even one that's just three hours long, actually increases productivity. (Some research suggests, by the way, that most workers are only productive for a few hours a day.)
For better or worse, technology might soon force us to reshape modern conceptions of the work week. That's primarily because A.I. is poised to threaten many human jobs, not necessarily just the menial ones, as SpaceX CEO Elon Musk suggested to the National Governors Association in 2017.
There certainly will be job disruption. Because what's going to happen is robots will be able to do everything better than us. . . I mean all of us.
In November, for instance, Lexus released the first TV commercial scripted by an A.I., suggesting that a fraction of the marketing industry could someday be replaced by algorithms that use vast amounts of consumer data to build advertisements. That might take a while. But robots are already replacing other human jobs, such as floor cleaners, and potentially burger flippers, at Walmart.
Still, it's worth noting that, for Musk at least, working less hours isn't necessarily a good thing, assuming you want to leave behind a legacy.
"There are way easier places to work, but nobody ever changed the world on 40 hours a week," Musk said, referring to his four companies, Tesla, SpaceX, The Boring Company, and Neuralink. Musk, who once said he was working 120 hours per week at Tesla, was asked on Twitter how many hours people should work if they want to change the world.
"Varies per person, but about 80 sustained, peaking above 100 at times," he said. "Pain level increases exponentially above 80."
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- The coronavirus pandemic may have a silver lining: It shows how insanely resourceful kids really are.
- Let Grow, a non-profit promoting independence as a critical part of childhood, ran an "Independence Challenge" essay contest for kids. Here are a few of the amazing essays that came in.
- Download Let Grow's free Independence Kit with ideas for kids.
Many Americans are being misled on serious scientific issues, and science journalists have to spend an inordinate amount of time debunking myths which seemingly never die.
Technique may enable speedy, on-demand design of softer, safer neural devices.
The brain is one of our most vulnerable organs, as soft as the softest tofu. Brain implants, on the other hand, are typically made from metal and other rigid materials that over time can cause inflammation and the buildup of scar tissue.
New research establishes an unexpected connection.
- A study provides further confirmation that a prolonged lack of sleep can result in early mortality.
- Surprisingly, the direct cause seems to be a buildup of Reactive Oxygen Species in the gut produced by sleeplessness.
- When the buildup is neutralized, a normal lifespan is restored.
We don't have to tell you what it feels like when you don't get enough sleep. A night or two of that can be miserable; long-term sleeplessness is out-and-out debilitating. Though we know from personal experience that we need sleep — our cognitive, metabolic, cardiovascular, and immune functioning depend on it — a lack of it does more than just make you feel like you want to die. It can actually kill you, according to study of rats published in 1989. But why?
A new study answers that question, and in an unexpected way. It appears that the sleeplessness/death connection has nothing to do with the brain or nervous system as many have assumed — it happens in your gut. Equally amazing, the study's authors were able to reverse the ill effects with antioxidants.
The study, from researchers at Harvard Medical School (HMS), is published in the journal Cell.
An unexpected culprit
The new research examines the mechanisms at play in sleep-deprived fruit flies and in mice — long-term sleep-deprivation experiments with humans are considered ethically iffy.
What the scientists found is that death from sleep deprivation is always preceded by a buildup of Reactive Oxygen Species (ROS) in the gut. These are not, as their name implies, living organisms. ROS are reactive molecules that are part of the immune system's response to invading microbes, and recent research suggests they're paradoxically key players in normal cell signal transduction and cell cycling as well. However, having an excess of ROS leads to oxidative stress, which is linked to "macromolecular damage and is implicated in various disease states such as atherosclerosis, diabetes, cancer, neurodegeneration, and aging." To prevent this, cellular defenses typically maintain a balance between ROS production and removal.
"We took an unbiased approach and searched throughout the body for indicators of damage from sleep deprivation," says senior study author Dragana Rogulja, admitting, "We were surprised to find it was the gut that plays a key role in causing death." The accumulation occurred in both sleep-deprived fruit flies and mice.
"Even more surprising," Rogulja recalls, "we found that premature death could be prevented. Each morning, we would all gather around to look at the flies, with disbelief to be honest. What we saw is that every time we could neutralize ROS in the gut, we could rescue the flies." Fruit flies given any of 11 antioxidant compounds — including melatonin, lipoic acid and NAD — that neutralize ROS buildups remained active and lived a normal length of time in spite of sleep deprivation. (The researchers note that these antioxidants did not extend the lifespans of non-sleep deprived control subjects.)
Image source: Tomasz Klejdysz/Shutterstock/Big Think
The study's tests were managed by co-first authors Alexandra Vaccaro and Yosef Kaplan Dor, both research fellows at HMS.
You may wonder how you compel a fruit fly to sleep, or for that matter, how you keep one awake. The researchers ascertained that fruit flies doze off in response to being shaken, and thus were the control subjects induced to snooze in their individual, warmed tubes. Each subject occupied its own 29 °C (84F) tube.
For their sleepless cohort, fruit flies were genetically manipulated to express a heat-sensitive protein in specific neurons. These neurons are known to suppress sleep, and did so — the fruit flies' activity levels, or lack thereof, were tracked using infrared beams.
Starting at Day 10 of sleep deprivation, fruit flies began dying, with all of them dead by Day 20. Control flies lived up to 40 days.
The scientists sought out markers that would indicate cell damage in their sleepless subjects. They saw no difference in brain tissue and elsewhere between the well-rested and sleep-deprived fruit flies, with the exception of one fruit fly.
However, in the guts of sleep-deprived fruit flies was a massive accumulation of ROS, which peaked around Day 10. Says Vaccaro, "We found that sleep-deprived flies were dying at the same pace, every time, and when we looked at markers of cell damage and death, the one tissue that really stood out was the gut." She adds, "I remember when we did the first experiment, you could immediately tell under the microscope that there was a striking difference. That almost never happens in lab research."
The experiments were repeated with mice who were gently kept awake for five days. Again, ROS built up over time in their small and large intestines but nowhere else.
As noted above, the administering of antioxidants alleviated the effect of the ROS buildup. In addition, flies that were modified to overproduce gut antioxidant enzymes were found to be immune to the damaging effects of sleep deprivation.
The research leaves some important questions unanswered. Says Kaplan Dor, "We still don't know why sleep loss causes ROS accumulation in the gut, and why this is lethal." He hypothesizes, "Sleep deprivation could directly affect the gut, but the trigger may also originate in the brain. Similarly, death could be due to damage in the gut or because high levels of ROS have systemic effects, or some combination of these."
The HMS researchers are now investigating the chemical pathways by which sleep-deprivation triggers the ROS buildup, and the means by which the ROS wreak cell havoc.
"We need to understand the biology of how sleep deprivation damages the body so that we can find ways to prevent this harm," says Rogulja.
Referring to the value of this study to humans, she notes,"So many of us are chronically sleep deprived. Even if we know staying up late every night is bad, we still do it. We believe we've identified a central issue that, when eliminated, allows for survival without sleep, at least in fruit flies."