Why monotasking is the new multitasking
Cross 'multi-tasking ninja' off your resume, it's out, say Stanford researchers and other cognitive experts. Here are three tips for transitioning back to single-tasking.
We know multi-tasking is bad for us: we just can't stop doing it. No matter how many times we hear that multitasking causes heightened mental stress, ruins memory and concentration more than smoking pot, and is literally impossible for our brains to do, we still do it.
“As much as people would like to believe otherwise, humans have finite neural resources that are depleted every time we switch between tasks," The New York Times reports. “That's why you feel tired at the end of the day. You've used them all up."
Multitasking is so pernicious because it doesn't feel like a myth. As Psychology Today explains, “When you multitask 'successfully,' you activate the reward mechanism in your brain which releases dopamine, the happy hormone. This dopamine rush makes you feel so good that you believe you're being effective and further encourages your multitasking habit." At first, multitasking appears to give us an enormous hit of that rush, and “that's why it's so hard to stop multitasking," Psychology Today says, “because you've conditioned your mind and body to feel that thrill."
In reality, multitasking splits our focus and gives us a false sense of accomplishment, making us “suckers for irrelevancy," as Stanford professor Clifford Nass put it in his 2009 study: “Everything distracts." ABC News correspondent Dan Harris concurs, as he told us:
In addition to all those deterrents, Psychology Today reports that multitasking can also “make you overly optimistic which means your [sic] less careful about the work you do and more likely to make mistakes." It also makes “the little information we do take in when we're multitasking more difficult to remember at a later stage." The Stanford research backs that up, with study co-author Eyal Ophir saying multitaskers “couldn't help thinking about the task they weren't doing. [They] are always drawing from all the information in front of them. They can't keep things separate in their minds," in a press release.
Those effects seem compounded in college students, since multitasking gave them lower GPAs according to this 2015 Iowa State University study. Neuroscientist and McGill University professor Daniel Levitin explains those myths to us here:
Given all this evidence, it's high time all of us heed the research and embrace monotasking. Monotasking — also known as unitasking or “single tasking," according to The Times — is not the same as mindfulness. Mindfulness cultivates awareness, a focus on the here and now. Monotasking is simply paying attention to, and completing, one task at a time.
If that sounds daunting to you, it can be. But don't worry: you can take baby steps to retrain your brain and reclaim your focus. Buffer co-founder Leo Widrich did just that with these 3 simple steps:
All those changes allowed him to maximize his neural energy and processes in a way that not only got his work done, but got it done faster and better than it did when he was multitasking.
If those steps are difficult for you to do at work, focus on the two biggest attention-busters: email and text messages. Inc recommends, "establishing an e-mail checking schedule" to avoid the temptation to check it every time you get an alert.
“Commit yourself to checking emails only three times a day, (maybe when you get into work in the morning, at lunchtime, and before leaving work at the end of the day)." They also suggest you “turn off texting notifications and choose specific times to check your phone as well" in order to minimize distractions during work.
If social media is your biggest distraction, there are ways to fix that, too. “You can get apps which block your social media (and even your email) except for certain times of the day," Psychology Today says. Here's a list from Mashable to get you started.
Whatever you decide, “make sure that you also take breaks in your unitasking because that's when your brain is at its most effective," Psychology Today says. One of the best things you can do during that break to recharge is meditate. Again, mindfulness is not the same thing as unitasking, but because mindfulness helps you focus on the present, it increases your focusing abilities. Here's Stanford neurosurgeon James Doty breaking down the process step by step:
Young people could even end up less anxiety-ridden, thanks to newfound confidence
- The coronavirus pandemic may have a silver lining: It shows how insanely resourceful kids really are.
- Let Grow, a non-profit promoting independence as a critical part of childhood, ran an "Independence Challenge" essay contest for kids. Here are a few of the amazing essays that came in.
- Download Let Grow's free Independence Kit with ideas for kids.
Philosophers like to present their works as if everything before it was wrong. Sometimes, they even say they have ended the need for more philosophy. So, what happens when somebody realizes they were mistaken?
Sometimes philosophers are wrong and admitting that you could be wrong is a big part of being a real philosopher. While most philosophers make minor adjustments to their arguments to correct for mistakes, others make large shifts in their thinking. Here, we have four philosophers who went back on what they said earlier in often radical ways.
New research establishes an unexpected connection.
- A study provides further confirmation that a prolonged lack of sleep can result in early mortality.
- Surprisingly, the direct cause seems to be a buildup of Reactive Oxygen Species in the gut produced by sleeplessness.
- When the buildup is neutralized, a normal lifespan is restored.
We don't have to tell you what it feels like when you don't get enough sleep. A night or two of that can be miserable; long-term sleeplessness is out-and-out debilitating. Though we know from personal experience that we need sleep — our cognitive, metabolic, cardiovascular, and immune functioning depend on it — a lack of it does more than just make you feel like you want to die. It can actually kill you, according to study of rats published in 1989. But why?
A new study answers that question, and in an unexpected way. It appears that the sleeplessness/death connection has nothing to do with the brain or nervous system as many have assumed — it happens in your gut. Equally amazing, the study's authors were able to reverse the ill effects with antioxidants.
The study, from researchers at Harvard Medical School (HMS), is published in the journal Cell.
An unexpected culprit
The new research examines the mechanisms at play in sleep-deprived fruit flies and in mice — long-term sleep-deprivation experiments with humans are considered ethically iffy.
What the scientists found is that death from sleep deprivation is always preceded by a buildup of Reactive Oxygen Species (ROS) in the gut. These are not, as their name implies, living organisms. ROS are reactive molecules that are part of the immune system's response to invading microbes, and recent research suggests they're paradoxically key players in normal cell signal transduction and cell cycling as well. However, having an excess of ROS leads to oxidative stress, which is linked to "macromolecular damage and is implicated in various disease states such as atherosclerosis, diabetes, cancer, neurodegeneration, and aging." To prevent this, cellular defenses typically maintain a balance between ROS production and removal.
"We took an unbiased approach and searched throughout the body for indicators of damage from sleep deprivation," says senior study author Dragana Rogulja, admitting, "We were surprised to find it was the gut that plays a key role in causing death." The accumulation occurred in both sleep-deprived fruit flies and mice.
"Even more surprising," Rogulja recalls, "we found that premature death could be prevented. Each morning, we would all gather around to look at the flies, with disbelief to be honest. What we saw is that every time we could neutralize ROS in the gut, we could rescue the flies." Fruit flies given any of 11 antioxidant compounds — including melatonin, lipoic acid and NAD — that neutralize ROS buildups remained active and lived a normal length of time in spite of sleep deprivation. (The researchers note that these antioxidants did not extend the lifespans of non-sleep deprived control subjects.)
Image source: Tomasz Klejdysz/Shutterstock/Big Think
The study's tests were managed by co-first authors Alexandra Vaccaro and Yosef Kaplan Dor, both research fellows at HMS.
You may wonder how you compel a fruit fly to sleep, or for that matter, how you keep one awake. The researchers ascertained that fruit flies doze off in response to being shaken, and thus were the control subjects induced to snooze in their individual, warmed tubes. Each subject occupied its own 29 °C (84F) tube.
For their sleepless cohort, fruit flies were genetically manipulated to express a heat-sensitive protein in specific neurons. These neurons are known to suppress sleep, and did so — the fruit flies' activity levels, or lack thereof, were tracked using infrared beams.
Starting at Day 10 of sleep deprivation, fruit flies began dying, with all of them dead by Day 20. Control flies lived up to 40 days.
The scientists sought out markers that would indicate cell damage in their sleepless subjects. They saw no difference in brain tissue and elsewhere between the well-rested and sleep-deprived fruit flies, with the exception of one fruit fly.
However, in the guts of sleep-deprived fruit flies was a massive accumulation of ROS, which peaked around Day 10. Says Vaccaro, "We found that sleep-deprived flies were dying at the same pace, every time, and when we looked at markers of cell damage and death, the one tissue that really stood out was the gut." She adds, "I remember when we did the first experiment, you could immediately tell under the microscope that there was a striking difference. That almost never happens in lab research."
The experiments were repeated with mice who were gently kept awake for five days. Again, ROS built up over time in their small and large intestines but nowhere else.
As noted above, the administering of antioxidants alleviated the effect of the ROS buildup. In addition, flies that were modified to overproduce gut antioxidant enzymes were found to be immune to the damaging effects of sleep deprivation.
The research leaves some important questions unanswered. Says Kaplan Dor, "We still don't know why sleep loss causes ROS accumulation in the gut, and why this is lethal." He hypothesizes, "Sleep deprivation could directly affect the gut, but the trigger may also originate in the brain. Similarly, death could be due to damage in the gut or because high levels of ROS have systemic effects, or some combination of these."
The HMS researchers are now investigating the chemical pathways by which sleep-deprivation triggers the ROS buildup, and the means by which the ROS wreak cell havoc.
"We need to understand the biology of how sleep deprivation damages the body so that we can find ways to prevent this harm," says Rogulja.
Referring to the value of this study to humans, she notes,"So many of us are chronically sleep deprived. Even if we know staying up late every night is bad, we still do it. We believe we've identified a central issue that, when eliminated, allows for survival without sleep, at least in fruit flies."
We must rethink the "chemical imbalance" theory of mental health.
- A new review found that withdrawal symptoms from antidepressants and antipsychotics can last for over a year.
- Side effects from SSRIs, SNRIs, and antipsychotics last longer than benzodiazepines like Valium or Prozac.
- The global antidepressant market is expected to reach $28.6 billion this year.