Emotional intelligence: What it is and do men or women have more of it
Daniel Goleman originated the theory of emotional intelligence. Here he describes the four domains that govern it, i.e. how we handle ourselves and our relationships.
Daniel Goleman is a psychologist, lecturer, and science journalist who has reported on the brain and behavioral sciences for The New York Times for many years. His 1995 book, Emotional Intelligence (Bantam Books) was on The New York Times bestseller list for a year and a half.
Goleman is also the author of Ecological Intelligence: How Knowing the Hidden Impacts of What We Buy Can Change Everything. The book argues that new information technologies will create “radical transparency,” allowing us to know the environmental, health, and social consequences of what we buy. As shoppers use point-of-purchase ecological comparisons to guide their purchases, market share will shift to support steady, incremental upgrades in how products are made – changing every thing for the better.
His latest book is Altered Traits: Science Reveals How Meditation Changes Your Mind, Brain, and Body, which he has co-authored with Richard Davidson reveals the science of what meditation can really do for us, as well as exactly how to get the most out of it.
Question: What is emotional intelligence?
Daniel Goleman: Emotional intelligence refers to how well we handle ourselves and our relationships, the 4 domains. Self-awareness, knowing what we’re feeling, why we’re feeling it, which is a basis of, for example, good intuition, good decision-making. Also, it’s a moral compass. Say, in part, is self-management, which means handling your distressing emotions in effective ways so that they don’t cripple you, they don’t get in the way of what you’re doing, and yet, attuning them… to them when you need to so that you learn what you must. Every emotion has a function. Also, [marshalling] positive emotions, getting ourselves, you know, involved, enthused about what we’re doing, aligning our actions with our passions. The third is empathy, knowing what someone else is feeling. And the fourth is putting that altogether in skilled relationship. So that’s what I mean by emotional intelligence. There’re many definitions out there. The part of the brain, it turns out, that supports emotional and social intelligence is actually the last circuitry of the brain to become anatomically mature. And because the neuroplasticity of the brain shapes itself according to repeated experiences, so my argument is, hey, we should be teaching kids regularly overtime, in a systematic way, self-awareness, self-management, empathy, and social skill. In fact, there, now, enough programs and they’ve been around enough in schools that they’re about to publish a huge meta analysis, looking at hundreds of schools and kids that had the program versus those that don’t. Guess what? All anti-social behavior, you know, disruption in class, find that… it goes down 10%. Pro-social behavior, liking school, well-behave, up 10%. Academic achievement scores, up 11%. So it really pays. Executive function, which is mediated by the prefrontal lobe, both helps you manage your emotions and helps you pay attention. So as kids learn these skills, they also learn learning… basic learning skills. I think that the fact that that was an argument was one thing that caught people’s attention. Then, there was a little chapter on… called managing with heart, which argued that leaders who were sons of a bitch were actually defeating the company’s own mission. And I think that made a lot of people happy because they work for people like that. I don’t know… Some people gave it to other people because they thought they needed help in this domain. I’m sure there’re a zillion reasons why people like the book.
Question: Are we becoming more emotionally intelligent?
Daniel Goleman: I hope more. I know IQ has been going up for a hundred years as children encounter more sophisticated cognitive environment as they grow. I don’t know that we’re becoming more emotionally intelligent. I like to hope we would but I think that the number of intergroup wars going on, the intergroup hatred going on, the, you know, levels of familial abuse, in other words, indicators of emotions out of control in dangerous ways don’t look that great, which is why I’m a very strong proponent of getting these social, emotional learning programs in every school worldwide.
Question: Are women more emotionally intelligent than men?
Daniel Goleman: Well, I get asked that question in a different way, which is, are women more emotionally intelligent than men? And you have to remember that emotional intelligence is a range of abilities, self-awareness, emotional self-management, empathy, social skills. Women tend to be better than men on average at empathy, particularly emotional empathy, sensing in the moment how the other person is feeling and also, at social skills, at keeping things feeling good between people in a group. Men, on the other hand, tend to be better on average at self-confidence, particularly in group, and at managing distressing emotions. But what’s very interesting is if you look at leaders who were in the top 10%, there’s no difference between the men and the women on any of those variables. In other words, you have a whole human being. So I would say that on average, there probably are differences men and women in this domain of ability. But as people develop their skills, as people become more effective, they pick up strengths in areas that they need.
Question: What cultures have the highest emotional intelligence?
Daniel Goleman: Well, I think that emotional intelligence as a universal but it looks different in different places. You know, Japan has a very rigid set of rules of social interaction, lots of subtleties. Americans typically blunder into the Japanese system, don’t get what’s going on. And, you know, it’s embarrassing but they wouldn’t recognize, necessarily, emotional intelligence in Japanese setting. Brazil is a very different culture. It’s very outgoing, you know, kind of like an Italian culture. And so, it will look different there but I think the fundamentals are the same.
Emotional intelligence is a range of abilities, self-awareness, emotional self-management, empathy, social skills. Women tend to be better than men on average at empathy, particularly emotional empathy, sensing in the moment how the other person is feeling and also, at social skills, at keeping things feeling good between people in a group. Men, on the other hand, tend to be better on average at self-confidence, particularly in groups, and at managing distressing emotions.
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She helped create CRISPR, a gene-editing technology that is changing the way we treat genetic diseases and even how we produce food.
This article was originally published on our sister site, Freethink.
Last year, Jennifer Doudna and Emmanuelle Charpentier became the first all-woman team to win the Nobel Prize in Chemistry for their work developing CRISPR-Cas9, the gene-editing technology. The technology was invented in 2012 — and nine years later, it's truly revolutionizing how we treat genetic diseases and even how we produce food.
CRISPR allows scientists to alter DNA by using proteins that are naturally found in bacteria. They use these proteins, called Cas9, to naturally fend off viruses, destroying the virus' DNA and cutting it out of their genes. CRISPR allows scientists to co-opt this function, redirecting the proteins toward disease-causing mutations in our DNA.
So far, gene-editing technology is showing promise in treating sickle cell disease and genetic blindness — and it could eventually be used to treat all sorts of genetic diseases, from cancer to Huntington's Disease.
The biotech revolution is just getting started — and CRISPR is leading the charge. We talked with Doudna about what we can expect from genetic engineering in the future.
This interview has been lightly edited and condensed for clarity.
Freethink: You've said that your journey to becoming a scientist had humble beginnings — in your teenage bedroom when you discovered The Double Helix by Jim Watson. Back then, there weren't a lot of women scientists — what was your breakthrough moment in realizing you could pursue this as a career?
Dr. Jennifer Doudna: There is a moment that I often think back to from high school in Hilo, Hawaii, when I first heard the word "biochemistry." A researcher from the UH Cancer Center on Oahu came and gave a talk on her work studying cancer cells.
I didn't understand much of her talk, but it still made a huge impact on me. You didn't see professional women scientists in popular culture at the time, and it really opened my eyes to new possibilities. She was very impressive.
I remember thinking right then that I wanted to do what she does, and that's what set me off on the journey that became my career in science.
CRISPR 101: Curing Sickle Cell, Growing Organs, Mosquito Makeovers | Jennifer Doudna | Big Think www.youtube.com
Freethink: The term "CRISPR" is everywhere in the media these days but it's a really complicated tool to describe. What is the one thing that you wish people understood about CRISPR that they usually get wrong?
Dr. Jennifer Doudna: People should know that CRISPR technology has revolutionized scientific research and will make a positive difference to their lives.
Researchers are gaining incredible new understanding of the nature of disease, evolution, and are developing CRISPR-based strategies to tackle our greatest health, food, and sustainability challenges.
Freethink: You previously wrote in Wired that this year, 2021, is going to be a big year for CRISPR. What exciting new developments should we be on the lookout for?
Dr. Jennifer Doudna: Before the COVID-19 pandemic, there were multiple teams around the world, including my lab and colleagues at the Innovative Genomics Institute, working on developing CRISPR-based diagnostics.
"Traits that we could select for using traditional breeding methods, that might take decades, we can now engineer precisely in a much shorter time."
DR. JENNIFER DOUDNA
When the pandemic hit, we pivoted our work to focus these tools on SARS-CoV-2. The benefit of these new diagnostics is that they're fast, cheap, can be done anywhere without the need for a lab, and they can be quickly modified to detect different pathogens. I'm excited about the future of diagnostics, and not just for pandemics.
We'll also be seeing more CRISPR applications in agriculture to help combat hunger, reduce the need for toxic pesticides and fertilizers, fight plant diseases and help crops adapt to a changing climate.
Traits that we could select for using traditional breeding methods, that might take decades, we can now engineer precisely in a much shorter time.
Freethink: Curing genetic diseases isn't a pipedream anymore, but there are still some hurdles to cross before we're able to say for certain that we can do this. What are those hurdles and how close do you think we are to crossing them?
Dr. Jennifer Doudna: There are people today, like Victoria Gray, who have been successfully treated for sickle cell disease. This is just the tip of the iceberg.
There are absolutely still many hurdles. We don't currently have ways to deliver genome-editing enzymes to all types of tissues, but delivery is a hot area of research for this very reason.
We also need to continue improving on the first wave of CRISPR therapies, as well as making them more affordable and accessible.
Freethink: Another big challenge is making this technology widely available to everyone and not just the really wealthy. You've previously said that this challenge starts with the scientists.
Dr. Jennifer Doudna: A sickle cell disease cure that is 100 percent effective but can't be accessed by most of the people in need is not really a full cure.
This is one of the insights that led me to found the Innovative Genomics Institute back in 2014. It's not enough to develop a therapy, prove that it works, and move on. You have to develop a therapy that actually meets the real-world need.
Too often, scientists don't fully incorporate issues of equity and accessibility into their research, and the incentives of the pharmaceutical industry tend to run in the opposite direction. If the world needs affordable therapy, you have to work toward that goal from the beginning.
Freethink: You've expressed some concern about the ethics of using CRISPR. Do you think there is a meaningful difference between enhancing human abilities — for example, using gene therapy to become stronger or more intelligent — versus correcting deficiencies, like Type 1 diabetes or Huntington's?
Dr. Jennifer Doudna: There is a meaningful distinction between enhancement and treatment, but that doesn't mean that the line is always clear. It isn't.
There's always a gray area when it comes to complex ethical issues like this, and our thinking on this is undoubtedly going to evolve over time.
What we need is to find an appropriate balance between preventing misuse and promoting beneficial innovation.
Freethink: What if it turns out that being physically stronger helps you live a longer life — if that's the case, are there some ways of improving health that we should simply rule out?
Dr. Jennifer Doudna: The concept of improving the "healthspan" of individuals is an area of considerable interest. Eliminating neurodegenerative disease will not only massively reduce suffering around the world, but it will also meaningfully increase the healthy years for millions of individuals.
"There is a meaningful distinction between enhancement and treatment, but that doesn't mean that the line is always clear. It isn't."
DR. JENNIFER DOUDNA
There will also be knock-on effects, such as increased economic output, but also increased impact on the planet.
When you think about increasing lifespans just so certain people can live longer, then not only do those knock-on effects become more central, you also have to ask who is benefiting and who isn't? Is it possible to develop this technology so the benefits are shared equitably? Is it environmentally sustainable to go down this road?
Freethink: Where do you see it going from here?
Dr. Jennifer Doudna: The bio revolution will allow us to create breakthroughs in treating not just a few but whole classes of previously unaddressed genetic diseases.
We're also likely to see genome editing play a role not just in climate adaptation, but in climate change solutions as well. There will be challenges along the way both expected and unexpected, but also great leaps in progress and benefits that will move society forward. It's an exciting time to be a scientist.
Freethink: If you had to guess, what is the first disease you think we are most likely to cure, in the real world, with CRISPR?
Dr. Jennifer Doudna: Because of the progress that has already been made, sickle cell disease and beta-thalassemia are likely to be the first diseases with a CRISPR cure, but we're closely following the developments of other CRISPR clinical trials for types of cancer, a form of congenital blindness, chronic infection, and some rare genetic disorders.
The pace of clinical trials is picking up, and the list will be longer next year.
A school lesson leads to more precise measurements of the extinct megalodon shark, one of the largest fish ever.
- A new method estimates the ancient megalodon shark was as long as 65 feet.
- The megalodon was one of the largest fish that ever lived.
- The new model uses the width of shark teeth to estimate its overall size.
A Florida student figured out a way to more accurately measure the size of one of the largest fish that ever lived – the extinct megalodon shark – and found that it was even larger than previously estimated.
The megalodon (officially named Otodus megalodon, which means "Big Tooth") lived between 3.6 and 23 million years ago and was thought to be about 34 feet long on average, reaching the maximum length of 60 feet. Now a new study puts that number at up to 65 feet (20 meters).
Homework assignment leads to a discovery
The study, published in Palaeontologia Electronica, used new equations extrapolated from the width of megalodon's teeth to make the improved estimates. The paper's lead author, Victor Perez, developed the revised methodology while he was a doctoral student at the Florida Museum of Natural History. He got the idea while teaching students, noticing a range of discrepancies in the results they were getting.
Students were supposed to calculate the size of megalodon based on the ancient fish's similarities to the modern great white shark. They utilized the commonly accepted method of linking the height of a shark's tooth to its total body length. As the press release from the Florida Museum of Natural History expounds, this method involves locating the anatomical position of a tooth in the shark's jaw, measuring the tooth "from the tip of the crown to the line where root and crown meet," and using that number in an appropriate equation.
But while carrying out calculations in this way, some of Perez's students thought the shark would have been just 40 feet long, while others were calculating 148 feet. Teeth located toward the back of the mouth were yielding the largest estimates.
"I was going around, checking, like, did you use the wrong equation? Did you forget to convert your units?" said Perez, currently the assistant curator of paleontology at the Calvert Marine Museum in Maryland. "But it very quickly became clear that it was not the students that had made the error. It was simply that the equations were not as accurate as we had predicted."
Found in North Carolina, these 46 fossils are the most complete set of megalodon teeth ever excavated.Credit: Jeff Gage/Florida Museum
The new approach
Perez's math exercise demonstrated that the equations in use since 2002 were generating different size estimates for the same shark based on which tooth was being measured. Because megalodon teeth are most often found as standalone fossils, Perez focused on a nearly complete set of teeth donated by a fossil collector to design a new approach.
Perez also had help from Teddy Badaut, an avocational paleontologist in France, who suggested using tooth width instead of height, which would be proportional to the length of its body. Another collaborator on the revised method was Ronny Maik Leder, then a postdoctoral researcher at the Florida Museum, who aided in the development of the new set of equations.
The research team analyzed the widths of fossil teeth that came from 11 individual sharks of five species, which included megalodon and modern great white sharks, and created a model that connects how wide a tooth was to the size of the jaw for each species.
"I was quite surprised that indeed no one had thought of this before," shared Leder, who is now director of the Natural History Museum in Leipzig, Germany. "The simple beauty of this method must have been too obvious to be seen. Our model was much more stable than previous approaches. This collaboration was a wonderful example of why working with amateur and hobby paleontologists is so important."
Why use teeth?
In general, almost nothing of the super-shark survived to this day, other than a few vertebrae and a large number of big teeth. The megalodon's skeleton was made of lightweight cartilage that decomposed after death. But teeth, with enamel that preserves very well, are "probably the most structurally stable thing in living organisms," Perez said. Considering that megalodons lost thousands of teeth during a lifetime, these are the best resources we have in trying to figure out information about these long-gone giants.
Researchers suggest megalodon's large jaws were very thick, made for grabbing prey and breaking its bones, exerting a bite force of up to 108,500 to 182,200 newtons.
Megalodon tooth compared to two great white shark teeth. Credit: Brocken Inaglory / Wikimedia.
Limitations of the new model
While the new model is better than previous methods, it's still far from perfect in precisely figuring out the sizes of animals which lived so long ago and left behind few if any full remains. Because individual sharks come in a variety of sizes, Perez warned that even their new estimates have an error range of about 10 feet when it comes to the largest animals.
Other ambiguities may affect the results, such as the width of the megalodon's jaw and the size of the gaps between its teeth, neither of which are accurately known. "There's still more that could be done, but that would probably require finding a complete skeleton at this point," Perez pointed out.
How did the megalodon go extinct?
Environmental changes that led to fluctuations in sea levels and disturbed ecosystems in the oceans likely led to the demise of these enormous ancient sharks. They were just too big to be sustained by diminishing food resources, says the ReefQuest Centre for Shark Research.
A 2018 study suggested that a supernova 2.6 million years ago hit Earth's atmosphere with so much cosmic energy that it resulted in climate change. The cosmic rays that included particles called muons might have caused a mass extinction of giant ocean animals ("the megafauna") that included the megalodon by causing mutations and cancer.
Scientists, led by Adrian Melott, professor emeritus of physics and astronomy at the University of Kansas, estimated that "the cancer rate would go up about 50 percent for something the size of a human — and the bigger you are, the worse it is. For an elephant or a whale, the radiation dose goes way up," as he explained in a press release.
We explore the history of blood types and how they are classified to find out what makes the Rh-null type important to science and dangerous for those who live with it.
- Fewer than 50 people worldwide have 'golden blood' — or Rh-null.
- Blood is considered Rh-null if it lacks all of the 61 possible antigens in the Rh system.
- It's also very dangerous to live with this blood type, as so few people have it.
Golden blood sounds like the latest in medical quackery. As in, get a golden blood transfusion to balance your tantric midichlorians and receive a free charcoal ice cream cleanse. Don't let the New-Agey moniker throw you. Golden blood is actually the nickname for Rh-null, the world's rarest blood type.
As Mosaic reports, the type is so rare that only about 43 people have been reported to have it worldwide, and until 1961, when it was first identified in an Aboriginal Australian woman, doctors assumed embryos with Rh-null blood would simply die in utero.
But what makes Rh-null so rare, and why is it so dangerous to live with? To answer that, we'll first have to explore why hematologists classify blood types the way they do.
A (brief) bloody history
Our ancestors understood little about blood. Even the most basic of blood knowledge — blood inside the body is good, blood outside is not ideal, too much blood outside is cause for concern — escaped humanity's grasp for an embarrassing number of centuries.
Absence this knowledge, our ancestors devised less-than-scientific theories as to what blood was, theories that varied wildly across time and culture. To pick just one, the physicians of Shakespeare's day believed blood to be one of four bodily fluids or "humors" (the others being black bile, yellow bile, and phlegm).
Handed down from ancient Greek physicians, humorism stated that these bodily fluids determined someone's personality. Blood was considered hot and moist, resulting in a sanguine temperament. The more blood people had in their systems, the more passionate, charismatic, and impulsive they would be. Teenagers were considered to have a natural abundance of blood, and men had more than women.
Humorism lead to all sorts of poor medical advice. Most famously, Galen of Pergamum used it as the basis for his prescription of bloodletting. Sporting a "when in doubt, let it out" mentality, Galen declared blood the dominant humor, and bloodletting an excellent way to balance the body. Blood's relation to heat also made it a go-to for fever reduction.
While bloodletting remained common until well into the 19th century, William Harvey's discovery of the circulation of blood in 1628 would put medicine on its path to modern hematology.
Soon after Harvey's discovery, the earliest blood transfusions were attempted, but it wasn't until 1665 that first successful transfusion was performed by British physician Richard Lower. Lower's operation was between dogs, and his success prompted physicians like Jean-Baptiste Denis to try to transfuse blood from animals to humans, a process called xenotransfusion. The death of human patients ultimately led to the practice being outlawed.4
The first successful human-to-human transfusion wouldn't be performed until 1818, when British obstetrician James Blundell managed it to treat postpartum hemorrhage. But even with a proven technique in place, in the following decades many blood-transfusion patients continued to die mysteriously.
Enter Austrian physician Karl Landsteiner. In 1901 he began his work to classify blood groups. Exploring the work of Leonard Landois — the physiologist who showed that when the red blood cells of one animal are introduced to a different animal's, they clump together — Landsteiner thought a similar reaction may occur in intra-human transfusions, which would explain why transfusion success was so spotty. In 1909, he classified the A, B, AB, and O blood groups, and for his work he received the 1930 Nobel Prize for Physiology or Medicine.
What causes blood types?
It took us a while to grasp the intricacies of blood, but today, we know that this life-sustaining substance consists of:
- Red blood cells — cells that carry oxygen and remove carbon dioxide throughout the body;
- White blood cells — immune cells that protect the body against infection and foreign agents;
- Platelets — cells that help blood clot; and
- Plasma — a liquid that carries salts and enzymes.6,7
Each component has a part to play in blood's function, but the red blood cells are responsible for our differing blood types. These cells have proteins* covering their surface called antigens, and the presence or absence of particular antigens determines blood type — type A blood has only A antigens, type B only B, type AB both, and type O neither. Red blood cells sport another antigen called the RhD protein. When it is present, a blood type is said to be positive; when it is absent, it is said to be negative. The typical combinations of A, B, and RhD antigens give us the eight common blood types (A+, A-, B+, B-, AB+, AB-, O+, and O-).
Blood antigen proteins play a variety of cellular roles, but recognizing foreign cells in the blood is the most important for this discussion.
Think of antigens as backstage passes to the bloodstream, while our immune system is the doorman. If the immune system recognizes an antigen, it lets the cell pass. If it does not recognize an antigen, it initiates the body's defense systems and destroys the invader. So, a very aggressive doorman.
While our immune systems are thorough, they are not too bright. If a person with type A blood receives a transfusion of type B blood, the immune system won't recognize the new substance as a life-saving necessity. Instead, it will consider the red blood cells invaders and attack. This is why so many people either grew ill or died during transfusions before Landsteiner's brilliant discovery.
This is also why people with O negative blood are considered "universal donors." Since their red blood cells lack A, B, and RhD antigens, immune systems don't have a way to recognize these cells as foreign and so leaves them well enough alone.
How is Rh-null the rarest blood type?
Let's return to golden blood. In truth, the eight common blood types are an oversimplification of how blood types actually work. As Smithsonian.com points out, "[e]ach of these eight types can be subdivided into many distinct varieties," resulting in millions of different blood types, each classified on a multitude of antigens combinations.
Here is where things get tricky. The RhD protein previously mentioned only refers to one of 61 potential proteins in the Rh system. Blood is considered Rh-null if it lacks all of the 61 possible antigens in the Rh system. This not only makes it rare, but this also means it can be accepted by anyone with a rare blood type within the Rh system.
This is why it is considered "golden blood." It is worth its weight in gold.
As Mosaic reports, golden blood is incredibly important to medicine, but also very dangerous to live with. If a Rh-null carrier needs a blood transfusion, they can find it difficult to locate a donor, and blood is notoriously difficult to transport internationally. Rh-null carriers are encouraged to donate blood as insurance for themselves, but with so few donors spread out over the world and limits on how often they can donate, this can also put an altruistic burden on those select few who agree to donate for others.
Some bloody good questions about blood types
A nurse takes blood samples from a pregnant woman at the North Hospital (Hopital Nord) in Marseille, southern France.
Photo by BERTRAND LANGLOIS / AFP
There remain many mysteries regarding blood types. For example, we still don't know why humans evolved the A and B antigens. Some theories point to these antigens as a byproduct of the diseases various populations contacted throughout history. But we can't say for sure.
In this absence of knowledge, various myths and questions have grown around the concept of blood types in the popular consciousness. Here are some of the most common and their answers.
Do blood types affect personality?
Japan's blood type personality theory is a contemporary resurrection of humorism. The idea states that your blood type directly affects your personality, so type A blood carriers are kind and fastidious, while type B carriers are optimistic and do their own thing. However, a 2003 study sampling 180 men and 180 women found no relationship between blood type and personality.
The theory makes for a fun question on a Cosmopolitan quiz, but that's as accurate as it gets.
Should you alter your diet based on your blood type?
Remember Galen of Pergamon? In addition to bloodletting, he also prescribed his patients to eat certain foods depending on which humors needed to be balanced. Wine, for example, was considered a hot and dry drink, so it would be prescribed to treat a cold. In other words, belief that your diet should complement your blood type is yet another holdover of humorism theory.
Created by Peter J. D'Adamo, the Blood Type Diet argues that one's diet should match one's blood type. Type A carriers should eat a meat-free diet of whole grains, legumes, fruits, and vegetables; type B carriers should eat green vegetables, certain meats, and low-fat dairy; and so on.
However, a study from the University of Toronto analyzed the data from 1,455 participants and found no evidence to support the theory. While people can lose weight and become healthier on the diet, it probably has more to do with eating all those leafy greens than blood type.
Are there links between blood types and certain diseases?
There is evidence to suggest that different blood types may increase the risk of certain diseases. One analysis suggested that type O blood decreases the risk of having a stroke or heart attack, while AB blood appears to increase it. With that said, type O carriers have a greater chance of developing peptic ulcers and skin cancer.
None of this is to say that your blood type will foredoom your medical future. Many factors, such as diet and exercise, hold influence over your health and likely to a greater extent than blood type.
What is the most common blood type?
In the United States, the most common blood type is O+. Roughly one in three people sports this type of blood. Of the eight well-known blood types, the least common is AB-. Only one in 167 people in the U.S. have it.
Do animals have blood types?
They most certainly do, but they are not the same as ours. This difference is why those 17th-century patients who thought, "Animal blood, now that's the ticket!" ultimately had their tickets punched. In fact, blood types are distinct between species. Unhelpfully, scientists sometimes use the same nomenclature to describe these different types. Cats, for example, have A and B antigens, but these are not the same A and B antigens found in humans.
Interestingly, xenotransfusion is making a comeback. Scientists are working to genetically engineer the blood of pigs to potentially produce human compatible blood.
Scientists are also looking into creating synthetic blood. If they succeed, they may be able to ease the current blood shortage, while also devising a way to create blood for rare blood type carriers. While this may make golden blood less golden, it would certainly make it easier to live with.* While antigens are typically proteins, they can be other molecules as well, such as polysaccharides.
Milgram's experiment is rightly famous, but does it show what we think it does?
- In the 1960s, Stanley Milgram was sure that good, law-abiding Americans would never be able to follow orders like the Germans in the Holocaust.
- His experiments proved him spectacularly wrong. They showed just how many of us are willing to do evil if only we're told to by an authority figure.
- Yet, parts of the experiment were set up in such a way that we should perhaps conclude something a bit more nuanced.
Holding a clipboard and wearing a lab coat makes you a very powerful person. Add in a lanyard and a confident voice, and you're pretty much in Ocean's Eleven.
Though we believe ourselves to be contrarians, most of us like to obey authority. We answer questions, help with any number of tasks, and obey commands unthinkingly. The vast majority of the time, this is relatively harmless and even requisite for a functioning society, but it can also lead humanity to very dark places.
It could happen here
As we've seen with Asch's experiments on conformity, the post-World War II community was determined to answer how and why the Holocaust took place. Just after the trial of Adolf Eichmann, the American media and public came to see German society as some special kind of monster in just how willing they were to follow orders unthinkingly, at odds with any sense of duty or morality.
Into this came Stanley Milgram. In 1961, Milgram set out a series of experiments to show, in his view, how the German people were more susceptible to authoritarianism than Americans. Milgram believed, as a lot of people did, that the American people would never be capable of such horrendous evil.
The experiment was to be set up in two stages: the first would be on American subjects, to gauge how far they would obey orders; the second would be on Germans, to prove how much they differed. The results stopped Milgram in his tracks.
Shock, shock, horror
Milgram wanted to ensure that his experiment involved as broad and diverse a group of people as possible. In addition to testing the American vs. German mindset, he wanted to see how much age, education, employment, and so on affected a person's willingness to obey orders.
So, the original 40 participants he gathered came from a wide spectrum of society, and each was told that they were to take part in a "memory test." They were to determine the extent to which punishment affects learning and the ability to memorize.
Milgram believed, as a lot of people did, that the American people would never be capable of such horrendous evil.
The experiment involved three people. First, there was the "experimenter," dressed in a lab coat, who gave instructions and prompts. Second, there was an actor who was the "learner." Third, there was the participant who thought that they were acting as the "teacher" in the memory test. The apparent experimental setup was that the learner had to match two words together after being taught them, and whenever they got the answer wrong, the teacher had to administer an electric shock. (The teachers (participants) were shocked as well to let them know what kind of pain the learner would experience.) At first, the shock was set at 15 volts.
The learner (actor) repeatedly made mistakes for each study, and the teacher was told to increase the voltage each time. A tape recorder was played that had the learner (apparently) make sounds as if in pain. As it went on, the learner would plead and beg for the shocks to stop. The teacher was told to increase the amount of voltage as punishment up to a level that was explicitly described as being fatal — not least because the learner was desperately saying he had a heart condition.
The question Milgram wanted to know: how far would his participants go?
Just obeying orders
The results were surprising. Sixty-five percent of the participants were willing to give a 450-volt shock described as lethal, and all administered a 300-volt shock described as traumatically painful. It should be repeated, this occurred despite the learner (actor) begging the teacher (participant) to stop.
In the studies that came after, in a variety of different setups, that 60 percent number came up again and again. They showed that roughly two out of three people would be willing to kill someone if told to by an authority figure. Milgram proved that all genders, ages, and nationalities were depressingly capable of inflicting incredible pain or worse on innocent people.
Major limitations in Milgram's experiment
Milgram took many steps to make sure that his experiment was rigorous and fair. He used the same tape recording of the "learner" screaming, begging, and pleading for all participants. He made sure the experimenters used only the same four prompts each time when the participants were reluctant or wanted to stop. He even made sure that he himself was not present at the experiment, lest he interfere with the procedure (something Phillip Zimbardo did not do).
But, does the Milgram experiment actually prove what we think it does?
First, the experimenters were permitted to remind the participants that they were not responsible for what they did and that the team would take full blame. This, of course, does not make the study any less shocking, but it does perhaps change the scope of the conclusions. Perhaps the experiment reveals more about our ability to surrender responsibility and our willingness simply to become a tool. The conclusion is still pretty depressing, but it shows what we are capable of when offered absolution rather than when simply following orders.
Second, the experiment took place in a single hour, with very little time either to deliberate or talk things over with someone. In most situations, like the Holocaust, the perpetrators had ample time (years) to reflect on their actions, and yet, they still chose to turn up every day. Milgram perhaps highlights only how far we'll go in the heat of the moment.
Finally, the findings do not tell the whole tale. The participants were not engaging in sadistic glee to shock the learner. They all showed signs of serious distress and anxiety, such as nervous laughing fits. Some even had seizures. These were not willing accomplices but participants essentially forced to act a certain way. (Since then, many scientists have argued that Milgram's experiment is hugely unethical.)
The power of authority
That all being said, there's a reason why Milgram's experiment stays with us today. Whether it's evolutionarily or socially drilled into us, it seems that humans are capable of doing terrible things, if only we are told to do so by someone in power — or, at the very least, when we don't feel responsible for the consequences.
One silver lining to Milgram is in how it can inoculate us against such drone-like behavior. It can help us to resist. Simply knowing how far we can be manipulated helps allow us to say, "No."